ME/CFS Evidence Browser — Classified Studies on Myalgic Encephalomyelitis

581–600 of 729 studies

E3 PreliminaryReviewPEM not requiredReview-Narrative

Mitochondria disease due to humidifier disinfectants: diagnostic criteria and its evidences.

Leem, Jong Han, Kim, Hwan-Cheol·Environmental analysis, health and toxicology·2020

In 2011, millions of people in South Korea were exposed to toxic chemicals in humidifier disinfectants, which caused widespread illness beyond just lung damage. Researchers found that these chemicals harm the mitochondria—the energy-producing parts of cells—leading to symptoms like chronic fatigue, muscle pain, exercise intolerance, and neurological problems similar to those seen in ME/CFS. This study reviews evidence that mitochondrial damage from chemical exposure may explain why victims developed multi-system health problems.

Evidence Atlas

Evidence browser

Mitochondria disease due to humidifier disinfectants: diagnostic criteria and its evidences.

Could Cognitive Behavioural Therapy Be an Effective Treatment for Long COVID and Post COVID-19 Fatigue Syndrome? Lessons from the Qure Study for Q-Fever Fatigue Syndrome.

Informatics Inference of Exercise-Induced Modulation of Brain Pathways Based on Cerebrospinal Fluid Micro-RNAs in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

An Isolated Complex V Inefficiency and Dysregulated Mitochondrial Function in Immortalized Lymphocytes from ME/CFS Patients.

Exercise alters brain activation in Gulf War Illness and Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

Altered muscle membrane potential and redox status differentiates two subgroups of patients with chronic fatigue syndrome.

Chronic fatigue syndrome: progress and possibilities.

Association of small-fiber polyneuropathy with three previously unassociated rare missense SCN9A variants.

Profile of circulating microRNAs in myalgic encephalomyelitis and their relation to symptom severity, and disease pathophysiology.

Exercise alters cerebellar and cortical activity related to working memory in phenotypes of Gulf War Illness.

Exercise-induce hyperalgesia, complement system and elastase activation in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome - a secondary analysis of experimental comparative studies.

[From neurasthenia to post-exertion disease: Evolution of the diagnostic criteria of chronic fatigue syndrome/myalgic encephalomyelitis].

Medically Documenting Disability in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) Cases.

Suggested pathology of systemic exertion intolerance disease: Impairment of the E3 subunit or crossover of swinging arms of the E2 subunit of the pyruvate dehydrogenase complex decreases regeneration of cofactor dihydrolipoic acid of the E2 subunit.

Understanding Myalgic Encephalomyelitis/Chronic Fatigue Syndrome and the Emerging Osteopathic Approach: A Narrative Review.

Exercise challenge alters Default Mode Network dynamics in Gulf War Illness.

Oxidative stress in exercise training: the involvement of inflammation and peripheral signals.

Understanding neuromuscular disorders in chronic fatigue syndrome.

Assessment of Post-Exertional Malaise (PEM) in Patients with Myalgic Encephalomyelitis (ME) and Chronic Fatigue Syndrome (CFS): A Patient-Driven Survey.

The DePaul Symptom Questionnaire-2: A Validation Study.

Curated Collections

Neuroinflammation Evidence

Metabolism & Mitochondria

Biomarkers

PEM-Specific Research