ME/CFS Evidence Browser — Classified Studies on Myalgic Encephalomyelitis

581–600 of 737 studies

E3 PreliminaryCommentaryPEM requiredEditorial

Pathophysiology of skeletal muscle disturbances in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS).

Wirth, Klaus J, Scheibenbogen, Carmen·Journal of translational medicine·2021

This study proposes that ME/CFS may be caused by a problem with how the body's nervous system controls blood vessels and muscle function. The researchers suggest that faulty beta-2 receptors (proteins that normally help regulate these systems) lead to poor blood flow in muscles, buildup of harmful substances, and damage to the energy-producing structures inside muscle cells. This creates a harmful cycle where even small amounts of activity can trigger the same problems again and again, explaining why patients experience crashes after exertion.

Evidence Atlas

Evidence browser

Pathophysiology of skeletal muscle disturbances in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS).

Caring for the Patient with Severe or Very Severe Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

Reduced Endothelial Function in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome-Results From Open-Label Cyclophosphamide Intervention Study.

Environmental accommodations for university students affected by Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS).

Cardiopulmonary responses to exercise in an individual with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome during long-term treatment with intravenous saline: A case study.

Mitochondria disease due to humidifier disinfectants: diagnostic criteria and its evidences.

Association of small-fiber polyneuropathy with three previously unassociated rare missense SCN9A variants.

Informatics Inference of Exercise-Induced Modulation of Brain Pathways Based on Cerebrospinal Fluid Micro-RNAs in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

Exercise alters cerebellar and cortical activity related to working memory in phenotypes of Gulf War Illness.

A SWATH-MS analysis of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome peripheral blood mononuclear cell proteomes reveals mitochondrial dysfunction.

Documenting disability in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS).

Chronic fatigue syndrome: progress and possibilities.

An Isolated Complex V Inefficiency and Dysregulated Mitochondrial Function in Immortalized Lymphocytes from ME/CFS Patients.

Exercise alters brain activation in Gulf War Illness and Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

Could Cognitive Behavioural Therapy Be an Effective Treatment for Long COVID and Post COVID-19 Fatigue Syndrome? Lessons from the Qure Study for Q-Fever Fatigue Syndrome.

Development of a conceptual framework to underpin a health-related quality of life outcome measure in paediatric chronic fatigue syndrome/myalgic encephalopathy (CFS/ME): prioritisation through card ranking.

Profile of circulating microRNAs in myalgic encephalomyelitis and their relation to symptom severity, and disease pathophysiology.

Altered muscle membrane potential and redox status differentiates two subgroups of patients with chronic fatigue syndrome.

Assessment of Post-Exertional Malaise (PEM) in Patients with Myalgic Encephalomyelitis (ME) and Chronic Fatigue Syndrome (CFS): A Patient-Driven Survey.

The DePaul Symptom Questionnaire-2: A Validation Study.

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Neuroinflammation Evidence

Metabolism & Mitochondria

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