ME/CFS Evidence Browser — Classified Studies on Myalgic Encephalomyelitis

961–980 of 1,205 studies

E3 PreliminaryMechanismPEM not requiredMechanistic

Possible role of oxidative stress and immunological activation in mouse model of chronic fatigue syndrome and its attenuation by olive extract.

Gupta, Amit, Vij, Garima, Chopra, Kanwaljit·Journal of neuroimmunology·2010

Researchers used mice to study whether oxidative stress (chemical damage in cells) and immune activation play a role in ME/CFS-like symptoms. They found that mice treated with immune-triggering substances showed increased fatigue and pain sensitivity, along with signs of cellular damage. When given olive extract, these mice showed improvement in fatigue, pain, and markers of cellular damage.

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Possible role of oxidative stress and immunological activation in mouse model of chronic fatigue syndrome and its attenuation by olive extract.

Activity in the hypothalamo-hypophyseal-adrenocortical system on experimental induction of chronic fatigue syndrome.

Inhibition of xenotropic murine leukemia virus-related virus by APOBEC3 proteins and antiviral drugs.

Immunological aspects of chronic fatigue syndrome.

A role for the body burden of aluminium in vaccine-associated macrophagic myofasciitis and chronic fatigue syndrome.

Kindling and Oxidative Stress as Contributors to Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

Postulated vasoactive neuropeptide immunopathology affecting the blood-brain/blood-spinal barrier in certain neuropsychiatric fatigue-related conditions: A role for phosphodiesterase inhibitors in treatment?

[Chronic fatigue syndrome: a psychoneuroimmunological perspective].

Immunological similarities between cancer and chronic fatigue syndrome: the common link to fatigue?

Autoantibodies to lens epithelium-derived growth factor/transcription co-activator P75 (LEDGF/P75) in children with chronic nonspecific complaints and with positive antinuclear antibodies.

Why myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) may kill you: disorders in the inflammatory and oxidative and nitrosative stress (IO&NS) pathways may explain cardiovascular disorders in ME/CFS.

[Immunologic disturbances can explain chronic fatigue syndrome. Biological findings point towards somatogenesis].

Transcriptional control of complement activation in an exercise model of chronic fatigue syndrome.

[Activity of hypotnalamic-pituitary-adrenal axis by induction of experimental chronic fatigue syndrom].

[Cellular and molecular mechanisms of interaction between the neuroendocrine and immune systems under chronic fatigue syndrome in experiment].

Inflammatory and oxidative and nitrosative stress pathways underpinning chronic fatigue, somatization and psychosomatic symptoms.

Astragalus membranaceus flavonoids (AMF) ameliorate chronic fatigue syndrome induced by food intake restriction plus forced swimming.

Clinical impact of B-cell depletion with the anti-CD20 antibody rituximab in chronic fatigue syndrome: a preliminary case series.

Curcumin, a polyphenolic antioxidant, attenuates chronic fatigue syndrome in murine water immersion stress model.

A gene signature for post-infectious chronic fatigue syndrome.

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Neuroinflammation Evidence

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