ME/CFS Evidence Browser — Classified Studies on Myalgic Encephalomyelitis

961–980 of 1,198 studies

E3 PreliminaryReviewPEM not requiredReview-Narrative

[Chronic fatigue syndrome: a psychoneuroimmunological perspective].

van Houdenhove, B, Heijnen, C J·Tijdschrift voor psychiatrie·2009

This review explores how stress, the immune system, and the brain interact in ME/CFS. The authors suggest that both physical and emotional stress may contribute to developing and maintaining the illness. While some research shows that people with ME/CFS have unusual stress responses and immune activity, the results haven't been fully consistent, indicating we still need more research to understand exactly what's happening.

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[Chronic fatigue syndrome: a psychoneuroimmunological perspective].

Inflammatory and oxidative and nitrosative stress pathways underpinning chronic fatigue, somatization and psychosomatic symptoms.

Clinical impact of B-cell depletion with the anti-CD20 antibody rituximab in chronic fatigue syndrome: a preliminary case series.

Autoantibodies to lens epithelium-derived growth factor/transcription co-activator P75 (LEDGF/P75) in children with chronic nonspecific complaints and with positive antinuclear antibodies.

Why myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) may kill you: disorders in the inflammatory and oxidative and nitrosative stress (IO&NS) pathways may explain cardiovascular disorders in ME/CFS.

Curcumin, a polyphenolic antioxidant, attenuates chronic fatigue syndrome in murine water immersion stress model.

Can sustained arousal explain the Chronic Fatigue Syndrome?

[Immunologic disturbances can explain chronic fatigue syndrome. Biological findings point towards somatogenesis].

Immunological aspects of chronic fatigue syndrome.

[Activity of hypotnalamic-pituitary-adrenal axis by induction of experimental chronic fatigue syndrom].

Kindling and Oxidative Stress as Contributors to Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

A role for the body burden of aluminium in vaccine-associated macrophagic myofasciitis and chronic fatigue syndrome.

Moderate exercise increases expression for sensory, adrenergic, and immune genes in chronic fatigue syndrome patients but not in normal subjects.

Chronic fatigue syndrome and complement activation.

Modulation of antigen-induced chronic fatigue in mouse model of water immersion stress by naringin, a polyphenolic antioxidant.

Transcriptional control of complement activation in an exercise model of chronic fatigue syndrome.

A gene signature for post-infectious chronic fatigue syndrome.

Contribution of proteomics to the study of the role of cytokeratins in disease and physiopathology.

Gene expression subtypes in patients with chronic fatigue syndrome/myalgic encephalomyelitis.

Possible role for early-life immune insult including developmental immunotoxicity in chronic fatigue syndrome (CFS) or myalgic encephalomyelitis (ME).

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