ME/CFS Evidence Browser — Classified Studies on Myalgic Encephalomyelitis

381–400 of 1,205 studies

E2 ModerateCohortPEM unclearCross-Sectional

Cytokines in the cerebrospinal fluids of patients with chronic fatigue syndrome/myalgic encephalomyelitis.

Peterson, D, Brenu, E W, Gottschalk, G et al.·Mediators of inflammation·2015

Researchers tested cerebrospinal fluid (the fluid around the brain and spinal cord) from ME/CFS patients and healthy controls to see if immune chemicals called cytokines were different. Out of 27 different immune chemicals tested, they found that only one—called IL-10—was lower in ME/CFS patients. This suggests that immune system problems in the brain and nervous system may play a role in ME/CFS symptoms.

Evidence Atlas

Evidence browser

Cytokines in the cerebrospinal fluids of patients with chronic fatigue syndrome/myalgic encephalomyelitis.

Role of polymorphisms of inducible nitric oxide synthase and endothelial nitric oxide synthase in idiopathic environmental intolerances.

Cytokine expression provides clues to the pathophysiology of Gulf War illness and myalgic encephalomyelitis.

Pathway-focused genetic evaluation of immune and inflammation related genes with chronic fatigue syndrome.

Sympathetic activation is associated with increased IL-6, but not CRP in the absence of obesity: lessons from postural tachycardia syndrome and obesity.

Serum Immune Proteins in Moderate and Severe Chronic Fatigue Syndrome/Myalgic Encephalomyelitis Patients.

Increased expression of activation antigens on CD8+ T lymphocytes in Myalgic Encephalomyelitis/chronic fatigue syndrome: inverse associations with lowered CD19+ expression and CD4+/CD8+ ratio, but no associations with (auto)immune, leaky gut, oxidative and nitrosative stress biomarkers.

Polymorphism in COMT is associated with IgG3 subclass level and susceptibility to infection in patients with chronic fatigue syndrome.

Plasma cytokine expression in adolescent chronic fatigue syndrome.

A new case definition of Neuro-Inflammatory and Oxidative Fatigue (NIOF), a neuroprogressive disorder, formerly known as chronic fatigue syndrome or Myalgic Encephalomyelitis: results of multivariate pattern recognition methods and external validation by neuro-immune biomarkers.

Longitudinal analysis of immune abnormalities in varying severities of Chronic Fatigue Syndrome/Myalgic Encephalomyelitis patients.

Distinct plasma immune signatures in ME/CFS are present early in the course of illness.

Chronic fatigue syndrome: Features of a population of patients from northern Italy.

Value-added reporting of antinuclear antibody testing by automated indirect immunofluorescence analysis.

Attenuation of autoimmune responses to oxidative specific epitopes, but not nitroso-adducts, is associated with a better clinical outcome in Myalgic Encephalomyelitis/chronic fatigue syndrome.

Evidence for the existence of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) with and without abdominal discomfort (irritable bowel) syndrome.

Dimensions of pure chronic fatigue: psychophysical, cognitive and biological correlates in the chronic fatigue syndrome.

Role of adaptive and innate immune cells in chronic fatigue syndrome/myalgic encephalomyelitis.

[FREQUENCY OF IMMUNOGLOBULIN E DEFICIENCY AMONG PATIENTS WITH IMMUNODEPENDENT DISORDERS].

Epitopes of microbial and human heat shock protein 60 and their recognition in myalgic encephalomyelitis.

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