ME/CFS Evidence Browser — Classified Studies on Myalgic Encephalomyelitis

141–160 of 210 studies

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Role of mitochondria, oxidative stress and the response to antioxidants in myalgic encephalomyelitis/chronic fatigue syndrome: A possible approach to SARS-CoV-2 'long-haulers'?

Wood, Emily, Hall, Katherine H, Tate, Warren·Chronic diseases and translational medicine·2021

This study reviews research on how mitochondria (the energy-producing centers in our cells) and oxidative stress (cellular damage from harmful molecules) may contribute to ME/CFS and long COVID symptoms. The authors examine whether antioxidant treatments could help restore energy production and reduce fatigue in these conditions. They highlight that many COVID-19 long-haulers develop symptoms that look very similar to ME/CFS, suggesting they may share underlying biological causes.

Evidence Atlas

Evidence browser

Role of mitochondria, oxidative stress and the response to antioxidants in myalgic encephalomyelitis/chronic fatigue syndrome: A possible approach to SARS-CoV-2 'long-haulers'?

Benefits of micronutrient supplementation on nutritional status, energy metabolism, and subjective wellbeing.

[Spinach and quinoa - prospective food sources of biologically active substances].

The effect of myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) severity on cellular bioenergetic function.

Substrate utilisation of cultured skeletal muscle cells in patients with CFS.

Protective Effect of Hemin Against Experimental Chronic Fatigue Syndrome in Mice: Possible Role of Neurotransmitters.

Human Herpesvirus-6 Reactivation, Mitochondrial Fragmentation, and the Coordination of Antiviral and Metabolic Phenotypes in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome.

Sugars and Sweeteners: Structure, Properties and In Silico Modeling.

Jerusalem artichoke (Helianthus tuberosus L.) as a medicinal plant and its natural products.

How Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) Progresses: The Natural History of ME/CFS.

Acute oxalate nephropathy due to high vitamin C doses and exocrine pancreatic insufficiency.

The IDO Metabolic Trap Hypothesis for the Etiology of ME/CFS.

Understanding neuromuscular disorders in chronic fatigue syndrome.

Genetic Predisposition for Immune System, Hormone, and Metabolic Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Pilot Study.

Severe posterior hypometabolism but normal perfusion in a patient with chronic fatigue syndrome/myalgic encephalomyelitis revealed by PET/MRI.

Astaxanthin: A Potential Mitochondrial-Targeted Antioxidant Treatment in Diseases and with Aging.

Mitochondrial complex activity in permeabilised cells of chronic fatigue syndrome patients using two cell types.

Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Induced by Repeated Forced Swimming in Mice.

Antifatigue Potential Activity of Sarcodon imbricatus in Acute Excise-Treated and Chronic Fatigue Syndrome in Mice via Regulation of Nrf2-Mediated Oxidative Stress.

[Electroacupuncture of "Zusanli" (ST 36) Raises Muscular Force by Adjusting AMPK/PGC-1 α Signaling in Rats with Chronic Fatigue Syndrome].

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Neuroinflammation Evidence

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