ME/CFS Evidence Browser — Classified Studies on Myalgic Encephalomyelitis

981–1000 of 1,205 studies

E3 PreliminaryMechanismPEM not requiredMechanistic

Modulation of antigen-induced chronic fatigue in mouse model of water immersion stress by naringin, a polyphenolic antioxidant.

Vij, Garima, Gupta, Amit, Chopra, Kanwaljit·Fundamental & clinical pharmacology·2009

Researchers tested whether naringin, a natural compound found in citrus fruits, could help reduce fatigue-like symptoms in mice exposed to immune challenges and stress. Mice treated with naringin showed improvements in their activity levels, pain sensitivity, and markers of cellular damage compared to untreated mice, suggesting that reducing oxidative stress might help manage fatigue symptoms.

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Modulation of antigen-induced chronic fatigue in mouse model of water immersion stress by naringin, a polyphenolic antioxidant.

Moderate exercise increases expression for sensory, adrenergic, and immune genes in chronic fatigue syndrome patients but not in normal subjects.

Chronic fatigue syndrome and complement activation.

Can sustained arousal explain the Chronic Fatigue Syndrome?

Unravelling intracellular immune dysfunctions in chronic fatigue syndrome: interactions between protein kinase R activity, RNase L cleavage and elastase activity, and their clinical relevance.

Chronic fatigue syndrome: characteristics and possible causes for its pathogenesis.

Neuroendocrine and immune network re-modeling in chronic fatigue syndrome: an exploratory analysis.

Gene profiling of patients with chronic fatigue syndrome/myalgic encephalomyelitis.

Chronic fatigue syndrome with autoantibodies--the result of an augmented adjuvant effect of hepatitis-B vaccine and silicone implant.

Intracellular immune dysfunction in myalgic encephalomyelitis/chronic fatigue syndrome: state of the art and therapeutic implications.

Gene expression subtypes in patients with chronic fatigue syndrome/myalgic encephalomyelitis.

Possible role for early-life immune insult including developmental immunotoxicity in chronic fatigue syndrome (CFS) or myalgic encephalomyelitis (ME).

Contribution of proteomics to the study of the role of cytokeratins in disease and physiopathology.

Evidence of inflammatory immune signaling in chronic fatigue syndrome: A pilot study of gene expression in peripheral blood.

Acute phase phospholipids related to the cardiolipin of mitochondria in the sera of patients with chronic fatigue syndrome (CFS), chronic Ciguatera fish poisoning (CCFP), and other diseases attributed to chemicals, Gulf War, and marine toxins.

Current research priorities in chronic fatigue syndrome/myalgic encephalomyelitis: disease mechanisms, a diagnostic test and specific treatments.

[Antinuclear antibodies in patients with chronic fatigue syndrome].

Normalization of the increased translocation of endotoxin from gram negative enterobacteria (leaky gut) is accompanied by a remission of chronic fatigue syndrome.

Chronic fatigue syndrome: inflammation, immune function, and neuroendocrine interactions.

Increased serum IgM antibodies directed against phosphatidyl inositol (Pi) in chronic fatigue syndrome (CFS) and major depression: evidence that an IgM-mediated immune response against Pi is one factor underpinning the comorbidity between both CFS and depression.

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